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10.1093/eurheartj/ehu353 http://scihub22266oqcxt.onion/10.1093/eurheartj/ehu353 C4810806!4810806 !25230814     free     free     free Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25230814 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Eur+Heart+J 2014 ; 35 (43 ): 3013-20, 3020a-3020d Nephropedia Template TP {{tp|p=25230814 |t=2014. Biomechanical factors in atherosclerosis: mechanisms and clinical implications |pdf=|usr=}}{{25230814 }} gab.com Text Biomechanical factors in atherosclerosis: mechanisms and clinical implications JO: Eur Heart J http://www.kidney.de/pget.php?&tw=1&pmid=25230814 #FOAvip Blood vessels are exposed to multiple mechanical forces that are exerted on the vessel wall (radial, circumferential and longitudinal forces) or on the endothelial surface (shear stress). The stresses and strains experienced by arteries influence the initiation of atherosclerotic lesions, which develop at regions of arteries that are exposed to complex blood flow. In addition, plaque progression and eventually plaque rupture is influenced by a complex interaction between biological and mechanical factors-mechanical forces regulate the cellular and molecular composition of plaques and, conversely, the composition of plaques determines their ability to withstand mechanical load. A deeper understanding of these interactions is essential for designing new therapeutic strategies to prevent lesion development and promote plaque stabilization. Moreover, integrating clinical imaging techniques with finite element modelling techniques allows for detailed examination of local morphological and biomechanical characteristics of atherosclerotic lesions that may be of help in prediction of future events. In this ESC Position Paper on biomechanical factors in atherosclerosis, we summarize the current 'state of the art' on the interface between mechanical forces and atherosclerotic plaque biology and identify potential clinical applications and key questions for future research. Twit Text FOAVip Biomechanical factors in atherosclerosis: mechanisms and clinical implications ????Eur Heart J http://www.kidney.de/pget.php?&tw=1&pmid=25230814 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25230814 #FOAvip English Wikipedia <ref>{{Cite pmid|25230814 }}</ref> Biomechanical factors in atherosclerosis: mechanisms and clinical implications #MMPMID25230814 Kwak BR ; Bäck M ; Bochaton-Piallat ML ; Caligiuri G ; Daemen MJ ; Davies PF ; Hoefer IE ; Holvoet P ; Jo H ; Krams R ; Lehoux S ; Monaco C ; Steffens S ; Virmani R ; Weber C ; Wentzel JJ ; Evans PC Eur Heart J 2014[Nov]; 35 (43 ): 3013-20, 3020a-3020d PMID25230814 show ga Blood vessels are exposed to multiple mechanical forces that are exerted on the vessel wall (radial, circumferential and longitudinal forces) or on the endothelial surface (shear stress). The stresses and strains experienced by arteries influence the initiation of atherosclerotic lesions, which develop at regions of arteries that are exposed to complex blood flow. In addition, plaque progression and eventually plaque rupture is influenced by a complex interaction between biological and mechanical factors-mechanical forces regulate the cellular and molecular composition of plaques and, conversely, the composition of plaques determines their ability to withstand mechanical load. A deeper understanding of these interactions is essential for designing new therapeutic strategies to prevent lesion development and promote plaque stabilization. Moreover, integrating clinical imaging techniques with finite element modelling techniques allows for detailed examination of local morphological and biomechanical characteristics of atherosclerotic lesions that may be of help in prediction of future events. In this ESC Position Paper on biomechanical factors in atherosclerosis, we summarize the current 'state of the art' on the interface between mechanical forces and atherosclerotic plaque biology and identify potential clinical applications and key questions for future research. |Apoptosis/physiology [MESH] |Arteries/*physiology [MESH] |Atherosclerosis/*physiopathology [MESH] |Biomarkers/metabolism [MESH] |Biomechanical Phenomena/physiology [MESH] |Cell Proliferation/physiology [MESH] |Cellular Senescence/physiology [MESH] |Disease Progression [MESH] |Endothelial Cells/physiology [MESH] |Endothelium, Vascular/physiology [MESH] |Homeostasis/physiology [MESH] |Humans [MESH] |Mechanoreceptors/physiology [MESH] |Plaque, Atherosclerotic/physiopathology [MESH] |Rupture, Spontaneous/physiopathology [MESH] |Signal Transduction/physiology [MESH] |Stress, Mechanical [MESH]Biomechanical factors in atherosclerosis: mechanisms and clinical impl(epmc).pdf DeepDyve Pubget Overpricing