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10.1128/AEM.02636-13 http://scihub22266oqcxt.onion/10.1128/AEM.02636-13 C3837759!3837759 !24038684     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24038684 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Appl+Environ+Microbiol 2013 ; 79 (23 ): 7116-21 Nephropedia Template TP {{tp|p=24038684 |t=2013. Bacterial persister cell formation and dormancy |pdf=|usr=}}{{24038684 }} gab.com Text Bacterial persister cell formation and dormancy JO: Appl Environ Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=24038684 #FOAvip Bacterial cells may escape the effects of antibiotics without undergoing genetic change; these cells are known as persisters. Unlike resistant cells that grow in the presence of antibiotics, persister cells do not grow in the presence of antibiotics. These persister cells are a small fraction of exponentially growing cells (due to carryover from the inoculum) but become a significant fraction in the stationary phase and in biofilms (up to 1%). Critically, persister cells may be a major cause of chronic infections. The mechanism of persister cell formation is not well understood, and even the metabolic state of these cells is debated. Here, we review studies relevant to the formation of persister cells and their metabolic state and conclude that the best model for persister cells is still dormancy, with the latest mechanistic studies shedding light on how cells reach this dormant state. Twit Text FOAVip Bacterial persister cell formation and dormancy ????Appl Environ Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=24038684 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24038684 #FOAvip English Wikipedia <ref>{{Cite pmid|24038684 }}</ref> Bacterial persister cell formation and dormancy #MMPMID24038684 Wood TK ; Knabel SJ ; Kwan BW Appl Environ Microbiol 2013[Dec]; 79 (23 ): 7116-21 PMID24038684 show ga Bacterial cells may escape the effects of antibiotics without undergoing genetic change; these cells are known as persisters. Unlike resistant cells that grow in the presence of antibiotics, persister cells do not grow in the presence of antibiotics. These persister cells are a small fraction of exponentially growing cells (due to carryover from the inoculum) but become a significant fraction in the stationary phase and in biofilms (up to 1%). Critically, persister cells may be a major cause of chronic infections. The mechanism of persister cell formation is not well understood, and even the metabolic state of these cells is debated. Here, we review studies relevant to the formation of persister cells and their metabolic state and conclude that the best model for persister cells is still dormancy, with the latest mechanistic studies shedding light on how cells reach this dormant state. |*Bacterial Physiological Phenomena [MESH] |Anti-Bacterial Agents/pharmacology [MESH] |Bacteria/drug effects/*growth & development/*metabolism [MESH]Bacterial persister cell formation and dormancy (epmc).pdf DeepDyve Pubget Overpricing