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10.1038/nri3532 http://scihub22266oqcxt.onion/10.1038/nri3532 C5340150!5340150 !24064518     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24064518 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Nat+Rev+Immunol 2013 ; 13 (10 ): 722-37 Nephropedia Template TP {{tp|p=24064518 |t=2013. Autophagy in infection, inflammation and immunity |pdf=|usr=}}{{24064518 }} gab.com Text Autophagy in infection, inflammation and immunity JO: Nat Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=24064518 #FOAvip Autophagy is a fundamental eukaryotic pathway that has multiple effects on immunity. Autophagy is induced by pattern recognition receptors and, through autophagic adaptors, it provides a mechanism for the elimination of intracellular microorganisms. Autophagy controls inflammation through regulatory interactions with innate immune signalling pathways, by removing endogenous inflammasome agonists and through effects on the secretion of immune mediators. Moreover, autophagy contributes to antigen presentation and to T cell homeostasis, and it affects T cell repertoires and polarization. Thus, as we discuss in this Review, autophagy has multitiered immunological functions that influence infection, inflammation and immunity. Twit Text FOAVip Autophagy in infection, inflammation and immunity ????Nat Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=24064518 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24064518 #FOAvip English Wikipedia <ref>{{Cite pmid|24064518 }}</ref> Autophagy in infection, inflammation and immunity #MMPMID24064518 Deretic V ; Saitoh T ; Akira S Nat Rev Immunol 2013[Oct]; 13 (10 ): 722-37 PMID24064518 show ga Autophagy is a fundamental eukaryotic pathway that has multiple effects on immunity. Autophagy is induced by pattern recognition receptors and, through autophagic adaptors, it provides a mechanism for the elimination of intracellular microorganisms. Autophagy controls inflammation through regulatory interactions with innate immune signalling pathways, by removing endogenous inflammasome agonists and through effects on the secretion of immune mediators. Moreover, autophagy contributes to antigen presentation and to T cell homeostasis, and it affects T cell repertoires and polarization. Thus, as we discuss in this Review, autophagy has multitiered immunological functions that influence infection, inflammation and immunity. |*Immunity, Innate [MESH] |Adaptor Proteins, Signal Transducing/genetics/immunology [MESH] |Antigen Presentation [MESH] |Autophagy/genetics/*immunology [MESH] |Bacterial Infections/genetics/*immunology/metabolism [MESH] |Gene Expression Regulation [MESH] |Homeostasis [MESH] |Humans [MESH] |Inflammasomes/genetics/immunology [MESH] |Inflammation [MESH] |Protein Transport [MESH] |Receptors, Pattern Recognition/genetics/*immunology [MESH] |Signal Transduction [MESH] |T-Lymphocytes/immunology/microbiology/virology [MESH]Autophagy in infection, inflammation and immunity (epmc).pdf DeepDyve Pubget Overpricing