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10.1007/s10875-016-0254-9 http://scihub22266oqcxt.onion/10.1007/s10875-016-0254-9 C4891390!4891390 !26984755     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26984755 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26984755 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Clin+Immunol 2016 ; 36 Suppl 1 (ä): 18-24 Nephropedia Template TP {{tp|p=26984755 |t=2016. Autophagy in Plasma Cell Ontogeny and Malignancy |pdf=|usr=}}{{26984755 }} gab.com Text Autophagy in Plasma Cell Ontogeny and Malignancy JO: J Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26984755 #FOAvip Autophagy is a highly conserved pathway that recycles cytosolic material and organelles via lysosomal degradation. Once simplistically viewed as a non-selective survival strategy in dire straits, autophagy has emerged as a tightly regulated process ensuring organelle function, proteome plasticity, cell differentiation and tissue homeostasis, with key roles in physiology and disease. Selective target recognition, mediated by specific adapter proteins, enables autophagy to orchestrate highly specialized functions in innate and adaptive immunity. Among them, the shaping of plasma cells for sustainable antibody production through a negative control on their differentiation program. Moreover, memory B cells and long-lived plasma cells require autophagy to exist. Further, the plasma cell malignancy, multiple myeloma deploys abundant autophagy, essential for homeostasis, survival and drug resistance. Twit Text FOAVip Autophagy in Plasma Cell Ontogeny and Malignancy ????J Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=26984755 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26984755 #FOAvip English Wikipedia <ref>{{Cite pmid|26984755 }}</ref> Autophagy in Plasma Cell Ontogeny and Malignancy #MMPMID26984755 Milan E ; Fabbri M ; Cenci S J Clin Immunol 2016[May]; 36 Suppl 1 (ä): 18-24 PMID26984755 show ga Autophagy is a highly conserved pathway that recycles cytosolic material and organelles via lysosomal degradation. Once simplistically viewed as a non-selective survival strategy in dire straits, autophagy has emerged as a tightly regulated process ensuring organelle function, proteome plasticity, cell differentiation and tissue homeostasis, with key roles in physiology and disease. Selective target recognition, mediated by specific adapter proteins, enables autophagy to orchestrate highly specialized functions in innate and adaptive immunity. Among them, the shaping of plasma cells for sustainable antibody production through a negative control on their differentiation program. Moreover, memory B cells and long-lived plasma cells require autophagy to exist. Further, the plasma cell malignancy, multiple myeloma deploys abundant autophagy, essential for homeostasis, survival and drug resistance. |*Autophagy [MESH] |Animals [MESH] |Autophagosomes/immunology/metabolism [MESH] |B-Lymphocytes/cytology/immunology/metabolism/pathology [MESH] |Carrier Proteins/metabolism [MESH] |Cell Transformation, Neoplastic/genetics/immunology/metabolism [MESH] |Humans [MESH] |Immune System/cytology/immunology/metabolism [MESH] |Neoplasms, Plasma Cell/*etiology/*metabolism/pathology [MESH] |Plasma Cells/*immunology/*metabolism/pathology [MESH] |Protein Binding [MESH]Autophagy in Plasma Cell Ontogeny and Malignancy (epmc).pdf DeepDyve Pubget Overpricing