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10.1016/j.semnephrol.2016.03.005 http://scihub22266oqcxt.onion/10.1016/j.semnephrol.2016.03.005 C4920968!4920968 !27339383     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27339383 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Semin+Nephrol 2016 ; 36 (3 ): 174-88 Nephropedia Template TP {{tp|p=27339383 |t=2016. Autophagy and Tubular Cell Death in the Kidney |pdf=|usr=}}{{27339383 }} gab.com Text Autophagy and Tubular Cell Death in the Kidney JO: Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27339383 #FOAvip Many common renal insults such as ischemia and toxic injury primarily target the tubular epithelial cells, especially the highly metabolically active proximal tubular segment. Tubular epithelial cells are particularly dependent on autophagy to maintain homeostasis and respond to stressors. The pattern of autophagy in the kidney has a unique spatial and chronologic signature. Recent evidence has shown that there is complex cross-talk between autophagy and various cell death pathways. This review specifically discusses the interplay between autophagy and cell death in the renal tubular epithelia. It is imperative to review this topic because recent discoveries have improved our mechanistic understanding of the autophagic process and have highlighted its broad clinical applications, making autophagy a major target for drug development. Twit Text FOAVip Autophagy and Tubular Cell Death in the Kidney ????Semin Nephrol http://www.kidney.de/pget.php?&tw=1&pmid=27339383 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27339383 #FOAvip English Wikipedia <ref>{{Cite pmid|27339383 }}</ref> Autophagy and Tubular Cell Death in the Kidney #MMPMID27339383 Havasi A ; Dong Z Semin Nephrol 2016[May]; 36 (3 ): 174-88 PMID27339383 show ga Many common renal insults such as ischemia and toxic injury primarily target the tubular epithelial cells, especially the highly metabolically active proximal tubular segment. Tubular epithelial cells are particularly dependent on autophagy to maintain homeostasis and respond to stressors. The pattern of autophagy in the kidney has a unique spatial and chronologic signature. Recent evidence has shown that there is complex cross-talk between autophagy and various cell death pathways. This review specifically discusses the interplay between autophagy and cell death in the renal tubular epithelia. It is imperative to review this topic because recent discoveries have improved our mechanistic understanding of the autophagic process and have highlighted its broad clinical applications, making autophagy a major target for drug development. |*Apoptosis [MESH] |*Autophagy [MESH] |Acute Kidney Injury/*metabolism/pathology [MESH] |Humans [MESH] |Kidney Tubules/cytology/*metabolism/pathology [MESH]Autophagy and Tubular Cell Death in the Kidney (epmc).pdf DeepDyve Pubget Overpricing