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10.4161/23723556.2014.985913 http://scihub22266oqcxt.onion/10.4161/23723556.2014.985913 C4905302!4905302 !27308466     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27308466 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27308466 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mol+Cell+Oncol 2015 ; 2 (3 ): e985913 Nephropedia Template TP {{tp|p=27308466 |t=2015. Autophagy, cell death, and cancer |pdf=|usr=}}{{27308466 }} gab.com Text Autophagy, cell death, and cancer JO: Mol Cell Oncol http://www.kidney.de/pget.php?&tw=1&pmid=27308466 #FOAvip Autophagy is an evolutionarily conserved intracellular catabolic process that is used by all cells to degrade dysfunctional or unnecessary cytoplasmic components through delivery to the lysosome. Increasing evidence reveals that autophagic dysfunction is associated with human diseases, such as cancer. Paradoxically, although autophagy is well recognized as a cell survival process that promotes tumor development, it can also participate in a caspase-independent form of programmed cell death. Induction of autophagic cell death by some anticancer agents highlights the potential of this process as a cancer treatment modality. Here, we review our current understanding of the molecular mechanism of autophagy and the potential roles of autophagy in cell death, cancer development, and cancer treatment. Twit Text FOAVip Autophagy, cell death, and cancer ????Mol Cell Oncol http://www.kidney.de/pget.php?&tw=1&pmid=27308466 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27308466 #FOAvip English Wikipedia <ref>{{Cite pmid|27308466 }}</ref> Autophagy, cell death, and cancer #MMPMID27308466 Lin L ; Baehrecke EH Mol Cell Oncol 2015[Jul]; 2 (3 ): e985913 PMID27308466 show ga Autophagy is an evolutionarily conserved intracellular catabolic process that is used by all cells to degrade dysfunctional or unnecessary cytoplasmic components through delivery to the lysosome. Increasing evidence reveals that autophagic dysfunction is associated with human diseases, such as cancer. Paradoxically, although autophagy is well recognized as a cell survival process that promotes tumor development, it can also participate in a caspase-independent form of programmed cell death. Induction of autophagic cell death by some anticancer agents highlights the potential of this process as a cancer treatment modality. Here, we review our current understanding of the molecular mechanism of autophagy and the potential roles of autophagy in cell death, cancer development, and cancer treatment. äAutophagy, cell death, and cancer (epmc).pdf DeepDyve Pubget Overpricing