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10.1016/j.tim.2014.02.012 http://scihub22266oqcxt.onion/10.1016/j.tim.2014.02.012 C4041834!4041834 !24684968     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24684968 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Microbiol 2014 ; 22 (6 ): 317-25 Nephropedia Template TP {{tp|p=24684968 |t=2014. Anthrax lethal and edema toxins in anthrax pathogenesis |pdf=|usr=}}{{24684968 }} gab.com Text Anthrax lethal and edema toxins in anthrax pathogenesis JO: Trends Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=24684968 #FOAvip The pathophysiological effects resulting from many bacterial diseases are caused by exotoxins released by the bacteria. Bacillus anthracis, a spore-forming bacterium, is such a pathogen, causing anthrax through a combination of bacterial infection and toxemia. B. anthracis causes natural infection in humans and animals and has been a top bioterrorism concern since the 2001 anthrax attacks in the USA. The exotoxins secreted by B. anthracis use capillary morphogenesis protein 2 (CMG2) as the major toxin receptor and play essential roles in pathogenesis during the entire course of the disease. This review focuses on the activities of anthrax toxins and their roles in initial and late stages of anthrax infection. Twit Text FOAVip Anthrax lethal and edema toxins in anthrax pathogenesis ????Trends Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=24684968 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24684968 #FOAvip English Wikipedia <ref>{{Cite pmid|24684968 }}</ref> Anthrax lethal and edema toxins in anthrax pathogenesis #MMPMID24684968 Liu S ; Moayeri M ; Leppla SH Trends Microbiol 2014[Jun]; 22 (6 ): 317-25 PMID24684968 show ga The pathophysiological effects resulting from many bacterial diseases are caused by exotoxins released by the bacteria. Bacillus anthracis, a spore-forming bacterium, is such a pathogen, causing anthrax through a combination of bacterial infection and toxemia. B. anthracis causes natural infection in humans and animals and has been a top bioterrorism concern since the 2001 anthrax attacks in the USA. The exotoxins secreted by B. anthracis use capillary morphogenesis protein 2 (CMG2) as the major toxin receptor and play essential roles in pathogenesis during the entire course of the disease. This review focuses on the activities of anthrax toxins and their roles in initial and late stages of anthrax infection. |*Host-Pathogen Interactions [MESH] |Animals [MESH] |Anthrax/drug therapy/*microbiology [MESH] |Antigens, Bacterial/metabolism/*physiology [MESH] |Bacillus anthracis/metabolism/*pathogenicity [MESH] |Bacterial Toxins/metabolism [MESH] |Humans [MESH] |Mice [MESH] |Models, Biological [MESH]Anthrax lethal and edema toxins in anthrax pathogenesis (epmc).pdf DeepDyve Pubget Overpricing