Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1080/2159256X.2015.1093067 http://scihub22266oqcxt.onion/10.1080/2159256X.2015.1093067 C4755260!4755260!26942043     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26942043.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mob+Genet+Elements 2015 ; 5 (6): 81-5 Nephropedia Template TP {{tp|p=26942043|t=2015. Alu elements and DNA double-strand break repair |pdf=|usr=}}{{26942043}} gab.com Text Alu elements and DNA double-strand break repair JO: Mob Genet Elements http://www.kidney.de/pget.php?&tw=1&pmid=26942043 #FOAvip Alu elements represent one of the most common sources of homology and homeology in the human genome. Homeologous recombination between Alu elements represents a major form of genetic instability leading to deletions and duplications. Although these types of events have been studied extensively through genomic sequencing to assess the impact of Alu elements on disease mutations and genome evolution, the overall abundance of Alu elements in the genome often makes it difficult to assess the relevance of the Alu elements to specific recombination events. We recently reported a powerful new reporter gene system that allows the assessment of various cis and trans factors on the contribution of Alu elements to various forms of genetic instability. This allowed a quantitative measurement of the influence of mismatches on Alu elements and instability. It also confirmed that homeologous Alu elements are able to stimulate non-homologous end joining events in their vicinity. This appears to be dependent on portions of the mismatch repair pathway. We are now in a position to begin to unravel the complex influences of Alu density, mismatch and location with alterations of DNA repair processes in various tissues and tumors. Twit Text FOAVip Alu elements and DNA double-strand break repair ????Mob Genet Elements http://www.kidney.de/pget.php?&tw=1&pmid=26942043 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26942043 #FOAvip English Wikipedia <ref>{{Cite pmid|26942043}}</ref> Alu elements and DNA double-strand break repair #MMPMID26942043 White TB; Morales ME; Deininger PL Mob Genet Elements 2015[Nov]; 5 (6): 81-5 PMID26942043show ga Alu elements represent one of the most common sources of homology and homeology in the human genome. Homeologous recombination between Alu elements represents a major form of genetic instability leading to deletions and duplications. Although these types of events have been studied extensively through genomic sequencing to assess the impact of Alu elements on disease mutations and genome evolution, the overall abundance of Alu elements in the genome often makes it difficult to assess the relevance of the Alu elements to specific recombination events. We recently reported a powerful new reporter gene system that allows the assessment of various cis and trans factors on the contribution of Alu elements to various forms of genetic instability. This allowed a quantitative measurement of the influence of mismatches on Alu elements and instability. It also confirmed that homeologous Alu elements are able to stimulate non-homologous end joining events in their vicinity. This appears to be dependent on portions of the mismatch repair pathway. We are now in a position to begin to unravel the complex influences of Alu density, mismatch and location with alterations of DNA repair processes in various tissues and tumors. äAlu elements and DNA double-strand break repair (epmc).pdf DeepDyve Pubget Overpricing