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10.1016/j.gtc.2014.02.006 http://scihub22266oqcxt.onion/10.1016/j.gtc.2014.02.006 C4019969!4019969 !24813516     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24813516 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24813516 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Gastroenterol+Clin+North+Am 2014 ; 43 (2 ): 281-96 Nephropedia Template TP {{tp|p=24813516 |t=2014. Allergic mechanisms in eosinophilic esophagitis |pdf=|usr=}}{{24813516 }} gab.com Text Allergic mechanisms in eosinophilic esophagitis JO: Gastroenterol Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=24813516 #FOAvip Eosinophilic esophagitis is rapidly increasing in incidence. It is associated with food antigen-triggered, eosinophil-predominant inflammation, and the pathogenic mechanisms have many similarities to other chronic atopic diseases. Studies in animal models and from patients have suggested that allergic sensitization leads to food-specific IgE and T-helper lymphocyte type 2 cells, both of which seem to contribute to the pathogenesis along with basophils, mast cells, and antigen-presenting cells. In this review our current understandings of the allergic mechanisms that drive eosinophilic esophagitis are outlined, drawing from clinical and translational studies in humans as well as experimental animal models. Twit Text FOAVip Allergic mechanisms in eosinophilic esophagitis ????Gastroenterol Clin North Am http://www.kidney.de/pget.php?&tw=1&pmid=24813516 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24813516 #FOAvip English Wikipedia <ref>{{Cite pmid|24813516 }}</ref> Allergic mechanisms in eosinophilic esophagitis #MMPMID24813516 Wechsler JB ; Bryce PJ Gastroenterol Clin North Am 2014[Jun]; 43 (2 ): 281-96 PMID24813516 show ga Eosinophilic esophagitis is rapidly increasing in incidence. It is associated with food antigen-triggered, eosinophil-predominant inflammation, and the pathogenic mechanisms have many similarities to other chronic atopic diseases. Studies in animal models and from patients have suggested that allergic sensitization leads to food-specific IgE and T-helper lymphocyte type 2 cells, both of which seem to contribute to the pathogenesis along with basophils, mast cells, and antigen-presenting cells. In this review our current understandings of the allergic mechanisms that drive eosinophilic esophagitis are outlined, drawing from clinical and translational studies in humans as well as experimental animal models. |Eosinophilic Esophagitis/*immunology [MESH] |Humans [MESH]Allergic mechanisms in eosinophilic esophagitis (epmc).pdf DeepDyve Pubget Overpricing