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10.1097/01.mnh.0000447017.28852.52 http://scihub22266oqcxt.onion/10.1097/01.mnh.0000447017.28852.52 C4096690!4096690!24848936     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24848936&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24848936.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Opin+Nephrol+Hypertens 2014 ; 23 (4): 385-90 Nephropedia Template TP {{tp|p=24848936|t=2014. Acute and chronic kidney injury in nephrolithiasis |pdf=|usr=}}{{24848936}} gab.com Text Acute and chronic kidney injury in nephrolithiasis JO: Curr Opin Nephrol Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=24848936 #FOAvip Purpose of review: Nephrolithiasis is a common systemic disease associated with both acute kidney injury (AKI) and chronic kidney disease (CKD). The purpose of this review is to discuss recent publications on nephrolithiasis-associated kidney damage, with an emphasis on AKI. Recent findings: Nephrolithiasis is not a common cause of adult AKI (1?2% of cases), although it may be a more important factor in young children (up to 30%). The primary mechanism of nephrolithiasis-associated AKI is obstructive nephropathy, and factors at presentation with obstructive uropathy predict the likelihood of long-term renal recovery. Crystalline nephropathy is another potential pathway in certain circumstances that is often associated with a worse outcome. Recent studies have elucidated additional pathways whereby calcium oxalate crystals can cause acute injury, implicating innate immunity and intracellular inflammasome pathways. Several large cohort studies have demonstrated an independent association of nephrolithiasis with CKD and ESRD, although the effect size is modest. Urologic comorbidities, urinary infection, and shared underlying risk factors (e.g., diabetes, hypertension) all impact nephrolithiasis-associated CKD risk. Summary: Obstructive nephropathy and crystalline nephropathy both contribute to nephrolithiasis-associated AKI, although the latter appears to have a worse prognosis. Nephrolithiasis is an independent albeit small risk factor for CKD. Further study is needed to clarify the incidence and mechanisms of nephrolithiasis-associated AKI, and the relationship between nephrolithiasis-associated AKI and CKD. Twit Text FOAVip Acute and chronic kidney injury in nephrolithiasis ????Curr Opin Nephrol Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=24848936 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24848936 #FOAvip English Wikipedia <ref>{{Cite pmid|24848936}}</ref> Acute and chronic kidney injury in nephrolithiasis #MMPMID24848936 Tang X; Lieske JC Curr Opin Nephrol Hypertens 2014[Jul]; 23 (4): 385-90 PMID24848936show ga Purpose of review: Nephrolithiasis is a common systemic disease associated with both acute kidney injury (AKI) and chronic kidney disease (CKD). The purpose of this review is to discuss recent publications on nephrolithiasis-associated kidney damage, with an emphasis on AKI. Recent findings: Nephrolithiasis is not a common cause of adult AKI (1?2% of cases), although it may be a more important factor in young children (up to 30%). The primary mechanism of nephrolithiasis-associated AKI is obstructive nephropathy, and factors at presentation with obstructive uropathy predict the likelihood of long-term renal recovery. Crystalline nephropathy is another potential pathway in certain circumstances that is often associated with a worse outcome. Recent studies have elucidated additional pathways whereby calcium oxalate crystals can cause acute injury, implicating innate immunity and intracellular inflammasome pathways. Several large cohort studies have demonstrated an independent association of nephrolithiasis with CKD and ESRD, although the effect size is modest. Urologic comorbidities, urinary infection, and shared underlying risk factors (e.g., diabetes, hypertension) all impact nephrolithiasis-associated CKD risk. Summary: Obstructive nephropathy and crystalline nephropathy both contribute to nephrolithiasis-associated AKI, although the latter appears to have a worse prognosis. Nephrolithiasis is an independent albeit small risk factor for CKD. Further study is needed to clarify the incidence and mechanisms of nephrolithiasis-associated AKI, and the relationship between nephrolithiasis-associated AKI and CKD. äAcute and chronic kidney injury in nephrolithiasis (epmc).pdf DeepDyve Pubget Overpricing