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10.18632/oncotarget.19167 http://scihub22266oqcxt.onion/10.18632/oncotarget.19167 C5687692!5687692 !29179522     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29179522 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Oncotarget 2017 ; 8 (51 ): 89315-89325 Nephropedia Template TP {{tp|p=29179522 |t=2017. Acetylation and deacetylation in cancer stem-like cells |pdf=|usr=}}{{29179522 }} gab.com Text Acetylation and deacetylation in cancer stem-like cells JO: Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=29179522 #FOAvip Cancer stem-like cell (CSC) model has been established to investigate the underlying mechanisms of tumor initiation and progression. The imbalance between acetylation and deacetylation of histone or non-histone proteins, one of the important epigenetic modification processes, is closely associated with a wide variety of diseases including cancer. Acetylation and deacetylation are involved in various stemness-related signal pathways and drive the regulation of self-renewal and differentiation in normal developmental processes. Therefore, it is critical to explore their role in the maintenance of cancer stem-like cell traits. Here, we will review the extensive dysregulations of acetylation found in cancers and summarize their functional roles in sustaining CSC-like properties. Additionally, the use of deacetyltransferase inhibitors as an effective therapeutic strategy against CSCs is also discussed. Twit Text FOAVip Acetylation and deacetylation in cancer stem-like cells ????Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=29179522 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29179522 #FOAvip English Wikipedia <ref>{{Cite pmid|29179522 }}</ref> Acetylation and deacetylation in cancer stem-like cells #MMPMID29179522 Liu N ; Li S ; Wu N ; Cho KS Oncotarget 2017[Oct]; 8 (51 ): 89315-89325 PMID29179522 show ga Cancer stem-like cell (CSC) model has been established to investigate the underlying mechanisms of tumor initiation and progression. The imbalance between acetylation and deacetylation of histone or non-histone proteins, one of the important epigenetic modification processes, is closely associated with a wide variety of diseases including cancer. Acetylation and deacetylation are involved in various stemness-related signal pathways and drive the regulation of self-renewal and differentiation in normal developmental processes. Therefore, it is critical to explore their role in the maintenance of cancer stem-like cell traits. Here, we will review the extensive dysregulations of acetylation found in cancers and summarize their functional roles in sustaining CSC-like properties. Additionally, the use of deacetyltransferase inhibitors as an effective therapeutic strategy against CSCs is also discussed. äAcetylation and deacetylation in cancer stem-like cells (epmc).pdf DeepDyve Pubget Overpricing