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10.14218/JCTH.2015.00052 http://scihub22266oqcxt.onion/10.14218/JCTH.2015.00052 C4913076!4913076 !27350943     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27350943 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Clin+Transl+Hepatol 2016 ; 4 (2 ): 131-42 Nephropedia Template TP {{tp|p=27350943 |t=2016. Acetaminophen-Induced Hepatotoxicity: a Comprehensive Update |pdf=|usr=}}{{27350943 }} gab.com Text Acetaminophen-Induced Hepatotoxicity: a Comprehensive Update JO: J Clin Transl Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=27350943 #FOAvip Hepatic injury and subsequent hepatic failure due to both intentional and non-intentional overdose of acetaminophen (APAP) has affected patients for decades, and involves the cornerstone metabolic pathways which take place in the microsomes within hepatocytes. APAP hepatotoxicity remains a global issue; in the United States, in particular, it accounts for more than 50% of overdose-related acute liver failure and approximately 20% of the liver transplant cases. The pathophysiology, disease course and management of acute liver failure secondary to APAP toxicity remain to be precisely elucidated, and adverse patient outcomes with increased morbidity and mortality continue to occur. Although APAP hepatotoxicity follows a predictable timeline of hepatic failure, its clinical presentation might vary. N-acetylcysteine (NAC) therapy is considered as the mainstay therapy, but liver transplantation might represent a life-saving procedure for selected patients. Future research focus in this field may benefit from shifting towards obtaining antidotal knowledge at the molecular level, with focus on the underlying molecular signaling pathways. Twit Text FOAVip Acetaminophen-Induced Hepatotoxicity: a Comprehensive Update ????J Clin Transl Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=27350943 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27350943 #FOAvip English Wikipedia <ref>{{Cite pmid|27350943 }}</ref> Acetaminophen-Induced Hepatotoxicity: a Comprehensive Update #MMPMID27350943 Yoon E ; Babar A ; Choudhary M ; Kutner M ; Pyrsopoulos N J Clin Transl Hepatol 2016[Jun]; 4 (2 ): 131-42 PMID27350943 show ga Hepatic injury and subsequent hepatic failure due to both intentional and non-intentional overdose of acetaminophen (APAP) has affected patients for decades, and involves the cornerstone metabolic pathways which take place in the microsomes within hepatocytes. APAP hepatotoxicity remains a global issue; in the United States, in particular, it accounts for more than 50% of overdose-related acute liver failure and approximately 20% of the liver transplant cases. The pathophysiology, disease course and management of acute liver failure secondary to APAP toxicity remain to be precisely elucidated, and adverse patient outcomes with increased morbidity and mortality continue to occur. Although APAP hepatotoxicity follows a predictable timeline of hepatic failure, its clinical presentation might vary. N-acetylcysteine (NAC) therapy is considered as the mainstay therapy, but liver transplantation might represent a life-saving procedure for selected patients. Future research focus in this field may benefit from shifting towards obtaining antidotal knowledge at the molecular level, with focus on the underlying molecular signaling pathways. äAcetaminophen-Induced Hepatotoxicity: a Comprehensive Update (epmc).pdf DeepDyve Pubget Overpricing