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NIH conference Epstein-Barr virus infections: biology, pathogenesis, and management Straus SE; Cohen JI; Tosato G; Meier JAnn Intern Med 1993[Jan]; 118 (1): 45-58Epstein-Barr virus (EBV) encodes genes that ensure its persistence in human B lymphocytes. Some of the genes encourage B-cell proliferation; others are poised to evade or defeat immune recognition. Immune restraints on the virus, however, are typically so effective that most infections are never symptomatic. In contrast, acute infectious mononucleosis, a self-limited lymphoproliferative illness, is common in adolescents and young adults. Unbridled proliferative illnesses arise when cellular immunity is grossly defective. Treatment of EBV-associated syndromes is largely supportive. Antiviral drugs have no proven role except in patients with oral hairy leukoplakia. Vaccine development is technically feasible but is not considered a high priority for developed nations.|*Herpesviridae Infections/immunology/microbiology/therapy[MESH]|*Herpesvirus 4, Human/genetics/physiology[MESH]|B-Lymphocytes/microbiology[MESH]|Cell Transformation, Neoplastic[MESH]|Cell Transformation, Viral[MESH]|Humans[MESH]|Infectious Mononucleosis/diagnosis/epidemiology/therapy[MESH]|Lymphoproliferative Disorders/immunology/microbiology[MESH] |
