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lüll Hepatitis E virus and neurologic disorders Kamar N; Bendall RP; Peron JM; Cintas P; Prudhomme L; Mansuy JM; Rostaing L; Keane F; Ijaz S; Izopet J; Dalton HREmerg Infect Dis 2011[Feb]; 17 (2): 173-9Information about the spectrum of disease caused by hepatitis E virus (HEV) genotype 3 is emerging. During 2004-2009, at 2 hospitals in the United Kingdom and France, among 126 patients with locally acquired acute and chronic HEV genotype 3 infection, neurologic complications developed in 7 (5.5%): inflammatory polyradiculopathy (n = 3), Guillain-Barre syndrome (n = 1), bilateral brachial neuritis (n = 1), encephalitis (n = 1), and ataxia/proximal myopathy (n = 1). Three cases occurred in nonimmunocompromised patients with acute HEV infection, and 4 were in immunocompromised patients with chronic HEV infection. HEV RNA was detected in cerebrospinal fluid of all 4 patients with chronic HEV infection but not in that of 2 patients with acute HEV infection. Neurologic outcomes were complete resolution (n = 3), improvement with residual neurologic deficit (n = 3), and no improvement (n = 1). Neurologic disorders are an emerging extrahepatic manifestation of HEV infection.|Acute Disease[MESH]|Adult[MESH]|Chronic Disease[MESH]|Female[MESH]|France[MESH]|Guillain-Barre Syndrome/physiopathology/*virology[MESH]|Hepatitis E virus/*pathogenicity[MESH]|Hepatitis E/*complications/virology[MESH]|Humans[MESH]|Male[MESH]|Middle Aged[MESH]|Nervous System Diseases/physiopathology/*virology[MESH]|United Kingdom[MESH] |