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lüll Host responses to Candida albicans: Th17 cells and mucosal candidiasis Conti HR; Gaffen SLMicrobes Infect 2010[Jul]; 12 (7): 518-27Candida albicans causes mucosal and disseminated candidiasis, which represent serious problems for the rapidly expanding immunocompromised population. Until recently, Th1-mediated immunity was thought to confer the primary protection, particularly for oral candidiasis. However, emerging data indicate that the newly-defined Th17 compartment appears to play the predominant role in mucosal candidiasis.|Adaptive Immunity[MESH]|Animals[MESH]|Candida albicans/*immunology[MESH]|Candidiasis/*immunology/metabolism[MESH]|Cell Line[MESH]|Host-Pathogen Interactions[MESH]|Humans[MESH]|Immunity, Innate[MESH]|Interleukin-17/*metabolism[MESH]|Interleukin-22[MESH]|Interleukin-23/metabolism[MESH]|Interleukins/metabolism[MESH]|Models, Animal[MESH]|Mucous Membrane/*immunology/metabolism/microbiology[MESH]|T-Lymphocytes, Helper-Inducer/*immunology[MESH] |