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lüll Interleukin-12 family members and type I interferons in Th17-mediated inflammatory disorders Goriely S; Cavoy R; Goldman MAllergy 2009[May]; 64 (5): 702-9Cytokines produced by antigen-presenting cells govern the fate of helper T-cell responses. Herein, we review the impact of interleukin (IL)-23 and IL-27 on the outcome of T-helper (Th) 17 cell responses and discuss their impact in the pathogenesis of T-cell-mediated inflammatory disorders of autoimmune or allergic origin. We then discuss how type I interferons might influence the course of autoimmune diseases by tipping the balance between IL-12 family members.|Animals[MESH]|Antigen-Presenting Cells/*immunology/metabolism[MESH]|Autoimmune Diseases/immunology/metabolism[MESH]|Autoimmunity/immunology[MESH]|Humans[MESH]|Hypersensitivity/*immunology/metabolism[MESH]|Inflammation/*immunology/metabolism[MESH]|Interferon Type I/*immunology/metabolism[MESH]|Interleukin-12/*immunology/metabolism[MESH]|Interleukin-17/immunology/metabolism[MESH]|Interleukin-23/immunology/metabolism[MESH]|T-Lymphocytes, Helper-Inducer/*immunology/metabolism[MESH] |