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lüll Brugada syndrome: recent advances and controversies Antzelevitch C; Nof ECurr Cardiol Rep 2008[Sep]; 10 (5): 376-83The Brugada syndrome, first described as a new clinical entity in 1992, is widely recognized today as a form of inherited sudden cardiac arrest. The past 16 years witnessed a progressive increase in the number of reported cases and a dramatic proliferation of articles serving to define the clinical, genetic, cellular, ionic, and molecular aspects of the disease. This article provides a brief overview of recent advances in our understanding of the clinical presentation and molecular and cellular mechanisms and an update of existing controversies.|*Brugada Syndrome/complications/diagnosis/genetics/therapy[MESH]|Anti-Arrhythmia Agents/therapeutic use[MESH]|Death, Sudden, Cardiac/etiology[MESH]|Defibrillators, Implantable[MESH]|Diagnosis, Differential[MESH]|Electrocardiography[MESH]|Humans[MESH]|Muscle Proteins/genetics[MESH]|NAV1.5 Voltage-Gated Sodium Channel[MESH]|Risk Factors[MESH]|Sodium Channels/genetics[MESH] |