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Disease-modifying mutations in familial hypertrophic cardiomyopathy: complexity from simplicity Hilfiker-Kleiner D; Knoll RCirculation 2008[Apr]; 117 (14): 1775-7�|*Disease Models, Animal[MESH]|*Mutation[MESH]|Animals[MESH]|Calcium Signaling[MESH]|Cardiomyopathy, Dilated/*genetics/pathology[MESH]|Cardiomyopathy, Hypertrophic, Familial/*genetics/physiopathology[MESH]|Fibrosis[MESH]|Heterozygote[MESH]|Humans[MESH]|Mice[MESH]|Mice, Mutant Strains[MESH]|Mutation, Missense[MESH]|Renin-Angiotensin System/physiology[MESH]|STAT3 Transcription Factor/physiology[MESH]|Sarcomeres/metabolism[MESH]|Signal Transduction[MESH]|Tachycardia, Ventricular/etiology[MESH]|Troponin I/*genetics[MESH]|Ventricular Myosins/*genetics/physiology[MESH] |