suck pdf from google scholar
unlimited free pdf from europmc18177599    free
PDF from PMC    free
html from PMC    free
suck pdf from library genesis
English Wikipedia

Nephropedia Template TP (

Twit Text


  • DeepDyve
  • Pubget Overpricing


  • lüll
  • Pathophysiology of psoriasis: recent advances on IL-23 and Th17 cytokines
  • Fitch E; Harper E; Skorcheva I; Kurtz SE; Blauvelt A
  • Curr Rheumatol Rep 2007[Dec]; 9 (6): 461-7
  • T helper (Th) 17 cells, a novel T-cell subset, have been implicated in the pathogenesis of psoriasis and other autoimmune inflammatory diseases. Interleukin (IL)-23 stimulates survival and proliferation of Th17 cells, and thus serves as a key master cytokine regulator for these diseases. In psoriasis, IL-23 is overproduced by dendritic cells and keratinocytes, and this cytokine stimulates Th17 cells within dermis to make IL-17A and IL-22. IL-22, in particular, drives keratinocyte hyperproliferation in psoriasis. Future targeting of these key cytokines is likely to lead to dramatic clinical improvement in patients with psoriasis. This review focuses on the numerous recent studies on the roles of IL-23 and Th17 cells in the pathogenesis of psoriasis.
  • |Dendritic Cells/immunology/metabolism/pathology[MESH]
  • |Humans[MESH]
  • |Interleukin-17/*immunology/metabolism[MESH]
  • |Interleukin-23/*immunology/metabolism[MESH]
  • |Keratinocytes/immunology/metabolism/pathology[MESH]
  • |Psoriasis/*immunology/metabolism/pathology[MESH]
  • |Signal Transduction[MESH]
  • |T-Lymphocyte Subsets/*immunology/metabolism/pathology[MESH]





  • *{{pmid18177599}}
    *<b>[http://www.kidney.de/mlpefetch.php?search=18177599 Pathophysiology of psoriasis: recent advances on IL-23 and Th17 cytokines ]</b> Curr Rheumatol Rep 2007; 9(6) ; 461-7 Fitch E; Harper E; Skorcheva I; Kurtz SE; Blauvelt A

        *18177599*

    Nephropedia PMID record

    Deutsche Wikipedia - im Artikel

    Hier den unten stehenden Textblock hineinkopieren

    Curr Rheumatol Rep

    461 6.9 2007