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lüll Lance-Adams syndrome: a report of two cases Zhang YX; Liu JR; Jiang B; Liu HQ; Ding MP; Song SJ; Zhang BR; Zhang H; Xu B; Chen HH; Wang ZJ; Huang JZJ Zhejiang Univ Sci B 2007[Oct]; 8 (10): 715-20Chronic post-hypoxic myoclonus, also known as Lance-Adams syndrome (LAS), is a rare complication of successful cardiopulmanry resuscitation often accompanied by action myoclonus and cerebellar ataxia. It is seen in patients who have undergone a cardiorespiratory arrest, regained consciousness afterwards, and then developed myoclonus days or weeks after the event. Worldwide, 122 cases have been reported in the literature so far, including 1 case of Chinese. Here we report 2 Chinese LAS patients with detailed neuroimagings. Cranial single photon emission computed tomography (SPECT) of patient 1, a 52-year-old woman, showed a mild hypoperfusion in her left temporal lobe, whereas patient 2, a 54-year-old woman, manifested a mild bilateral decrease of glucose metabolism in the frontal lobes and a mild to moderate decrease of the N-acetyl aspartate (NAA) peak in the bilateral hippocampi by cranial [(18)F]-fluorodeoxyglucose positron emission tomographic (PET) scan and cranial magnetic resonance spectroscopy (MRS), respectively. We also review the literature on the neuroimaging, pathogenesis, and treatment of LAS.|Cardiopulmonary Resuscitation/*adverse effects[MESH]|Cerebellar Ataxia/*diagnosis/*etiology[MESH]|Female[MESH]|Humans[MESH]|Hypoxia-Ischemia, Brain/*diagnosis/*etiology[MESH]|Middle Aged[MESH]|Myoclonus/*diagnosis/*etiology[MESH]|Syndrome[MESH] |