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lüll Mechanisms of eosinophilia in the pathogenesis of hypereosinophilic disorders Ackerman SJ; Bochner BSImmunol Allergy Clin North Am 2007[Aug]; 27 (3): 357-75The increased numbers of activated eosinophils in the blood and tissues that typically accompany hypereosinophilic disorders result from a variety of mechanisms. Exciting advances in translating discoveries achieved from mouse models and molecular strategies to the clinic have led to a flurry of new therapeutics specifically designed to target eosinophil-associated diseases. So far, this form of hypothesis testing in humans in vivo through pharmacology generally has supported the paradigms generated in vitro and in animal models, raising hopes that a spectrum of novel therapies soon may become available to help those who have eosinophil-associated diseases.|Animals[MESH]|Cell Degranulation[MESH]|Chemotactic Factors, Eosinophil/metabolism[MESH]|Cytokines/immunology/*metabolism[MESH]|Eosinophil Granule Proteins/immunology/*metabolism[MESH]|Eosinophils/*immunology/*physiology[MESH]|Humans[MESH]|Hypereosinophilic Syndrome/blood/immunology/*physiopathology[MESH]|Inflammation Mediators/immunology/*metabolism[MESH]|Leukopoiesis[MESH] |