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lüll Innate immune evasion by hepatitis C virus and West Nile virus Keller BC; Johnson CL; Erickson AK; Gale M JrCytokine Growth Factor Rev 2007[Oct]; 18 (5-6): 535-44Antiviral immunity in mammals involves several levels of surveillance and effector actions by host factors to detect viral pathogens, trigger alpha/beta interferon production, and to mediate innate defenses within infected cells. Our studies have focused on understanding how these processes are regulated during infection by hepatitis C virus (HCV) and West Nile virus (WNV). Both viruses are members of the Flaviviridae and are human pathogens, but they each mediate a very different disease and course of infection. Our results demonstrate common and unique innate immune interactions of each virus that govern antiviral immunity and demonstrate the central role of alpha/beta interferon immune defenses in controlling the outcome of infection.|Animals[MESH]|Hepacivirus/*pathogenicity[MESH]|Humans[MESH]|Immunity, Innate[MESH]|Interferons/*immunology[MESH]|West Nile virus/*pathogenicity[MESH] |