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lüll FOXP3 and NFAT: partners in tolerance Rudensky AY; Gavin M; Zheng YCell 2006[Jul]; 126 (2): 253-6Regulatory T cells suppress autoimmune responses to self-antigens. Recent studies, including one in this issue of Cell (Wu et al., 2006), suggest that the ability of T cells to choose between launching a productive immune response, functional inactivation, or developing into regulatory T cells depends upon the interplay of the key transcriptional regulators FOXP3 and NFAT.|*Immune Tolerance[MESH]|Amino Acid Sequence[MESH]|Amino Acid Substitution[MESH]|Autoimmunity[MESH]|Base Sequence[MESH]|Biomarkers/metabolism[MESH]|CD24 Antigen/immunology[MESH]|Crystallography, X-Ray[MESH]|Forkhead Transcription Factors/chemistry/genetics/*immunology[MESH]|Gene Expression Regulation/genetics/immunology[MESH]|Genes, Reporter[MESH]|Humans[MESH]|Hydrogen Bonding[MESH]|Interleukin-2/genetics/immunology[MESH]|Jurkat Cells[MESH]|Luciferases/metabolism[MESH]|Lymphocyte Activation[MESH]|Models, Biological[MESH]|Molecular Sequence Data[MESH]|NFATC Transcription Factors/chemistry/genetics/*immunology[MESH]|Protein Structure, Tertiary[MESH]|Receptors, Interleukin-2/immunology[MESH]|Sequence Homology, Amino Acid[MESH]|T-Lymphocytes, Regulatory/immunology[MESH] |