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lüll Pathogenesis of calcific aortic valve disease: a disease process comes of age (and a good deal more) O'Brien KDArterioscler Thromb Vasc Biol 2006[Aug]; 26 (8): 1721-8Over the past 10 to 15 years, calcific aortic valve disease, which includes aortic sclerosis and aortic stenosis, has come to be recognized as an active process, based on: (1) epidemiologic studies demonstrating associations of specific risk factors with increased prevalence or rate of progression of aortic valve disease; (2) identification, in valve lesions, of histopathologic features of chronic inflammation, lipoprotein deposition, renin-angiotensin system components, and molecular mediators of calcification; and (3) identification of cell-signaling pathways and genetic factors that may participate in valve disease pathogenesis. These studies will be reviewed and organized into a proposed global hypothesis for the pathogenesis of calcific aortic valve disease.|*Aortic Valve[MESH]|Animals[MESH]|Calcinosis/*etiology[MESH]|Heart Valve Diseases/*etiology[MESH]|Humans[MESH] |