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lüll Blocking IL-1 in systemic inflammation Dinarello CAJ Exp Med 2005[May]; 201 (9): 1355-9A growing number of systemic inflammatory diseases characterized in part by recurrent fevers, leukocytosis, anemia, and elevated acute phase proteins are linked to interleukin (IL)-1 activity since rapid and sustained resolution is observed upon specific blockade of IL-1 receptors. Rapid resolution of systemic and local inflammation is now also reported in systemic onset juvenile idiopathic arthritis (SoJIA). Loss of control of the secretion of IL-1beta might be a common mechanism explaining the aberrant activity of IL-1 in these diseases.|*Models, Biological[MESH]|Arthritis, Juvenile/drug therapy/etiology/*metabolism[MESH]|Humans[MESH]|Inflammation/etiology/*metabolism[MESH]|Interleukin 1 Receptor Antagonist Protein[MESH]|Interleukin-1/*metabolism[MESH]|Receptors, Interleukin-1/*antagonists & inhibitors[MESH]|Sialoglycoproteins/metabolism/therapeutic use[MESH]|Tumor Necrosis Factor-alpha/metabolism[MESH] |