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Cysteinyl leukotrienes in allergic inflammation: strategic target for therapy Busse W; Kraft MChest 2005[Apr]; 127 (4): 1312-26Systemically bioavailable leukotriene receptor antagonists (LTRAs) can reduce the essential components of allergic inflammation in allergic rhinitis (AR) and asthma by blocking cysteinyl leukotriene (CysLT) activity, resulting in a wide range of clinical effects. CysLTs, mediators, and modulators in the pathophysiology of asthma and AR are a key target for therapy because they modulate production of hemopoietic progenitor cells, survival and recruitment of eosinophils to inflamed tissue, activity of cytokines and chemokines, quantity of exhaled NO, smooth-muscle contraction, and proliferation of fibroblasts. The mechanism of action of LTRAs leads to their effects on systemic allergic inflammatory processes.|Adult[MESH]|Child[MESH]|Cysteine/*antagonists & inhibitors/*immunology[MESH]|Humans[MESH]|Hypersensitivity/*drug therapy/genetics/*immunology[MESH]|Inflammation Mediators/*antagonists & inhibitors/*immunology[MESH]|Leukotrienes/*immunology[MESH]|Phenotype[MESH]|Quality of Life[MESH]|Rhinitis, Allergic, Perennial/*drug therapy/genetics/*immunology[MESH] |
