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lüll Cardiovascular biology of the asymmetric dimethylarginine:dimethylarginine dimethylaminohydrolase pathway Vallance P; Leiper JArterioscler Thromb Vasc Biol 2004[Jun]; 24 (6): 1023-30An increasing number of reports indicate that endogenously produced inhibitors of nitric oxide synthase, particularly asymmetric dimethylarginine (ADMA), regulate nitric oxide generation in disease states. This article describes the biology of ADMA and the implications for cardiovascular physiology and pathophysiology.|*Cardiovascular Physiological Phenomena[MESH]|Amidohydrolases/antagonists & inhibitors/*metabolism[MESH]|Animals[MESH]|Arginine/*analogs & derivatives/*metabolism[MESH]|Cardiovascular Diseases/drug therapy/metabolism/*physiopathology[MESH]|Diabetes Mellitus/metabolism[MESH]|Endothelium, Vascular/metabolism/physiopathology[MESH]|Enzyme Inhibitors/therapeutic use[MESH]|Female[MESH]|Humans[MESH]|Mice[MESH]|Models, Biological[MESH]|Nitric Oxide Synthase/antagonists & inhibitors[MESH]|Nitric Oxide/metabolism[MESH]|Pre-Eclampsia/metabolism[MESH]|Pregnancy[MESH]|Protein-Arginine N-Methyltransferases/metabolism[MESH]|Renal Insufficiency/metabolism[MESH]|omega-N-Methylarginine/pharmacology[MESH] |