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  • (Patho)physiological implications of the novel epithelial Ca2+ channels TRPV5 and TRPV6
  • Nijenhuis T; Hoenderop JG; Nilius B; Bindels RJ
  • Pflugers Arch 2003[Jul]; 446 (4): 401-9
  • The epithelial Ca(2+) channels TRPV5 and TRPV6 constitute the apical Ca(2+) entry mechanism in active Ca(2+) (re)absorption. These two members of the superfamily of transient receptor potential (TRP) channels were cloned from the vitamin-D-responsive epithelia of kidney and small intestine and subsequently identified in other tissues such as bone, pancreas and prostate. These channels are regulated by vitamin D as exemplified in animal models of vitamin-D-deficiency rickets. In addition, the epithelial Ca(2+) channels might be involved in the multifactorial pathogenesis of disorders ranging from idiopathic hypercalciuria, stone disease and postmenopausal osteoporosis. This review highlights the emerging (patho)physiological implications of these epithelial Ca(2+) channels.
  • |Animals[MESH]
  • |Calcium Channels/*physiology[MESH]
  • |Calcium/*metabolism[MESH]
  • |Epithelial Cells/*physiology[MESH]
  • |Humans[MESH]
  • |Metabolic Diseases/metabolism/*physiopathology[MESH]
  • |TRPV Cation Channels[MESH]





  • *{{pmid12748856}}
    *<b>[http://www.kidney.de/mlpefetch.php?search=12748856 (Patho)physiological implications of the novel epithelial Ca2+ channels TRPV5 and TRPV6 ]</b> Pflugers Arch 2003; 446(4) ; 401-9 Nijenhuis T; Hoenderop JG; Nilius B; Bindels RJ

        *12748856*

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    Pflugers Arch

    401 4.446 2003