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Fibrosis regression induced by intravenous gammaglobulin treatment Amital H; Rewald E; Levy Y; Bar-Dayan Y; Manthorpe R; Engervall P; Sherer Y; Langevitz P; Shoenfeld YAnn Rheum Dis 2003[Feb]; 62 (2): 175-7OBJECTIVES: To review case histories of patients in whom fibrosis played a significant role in the pathogenesis of their disease, and to determine whether intravenous gammaglobulin (IVIg) contributed to the regression of their fibrotic condition. METHODS: Eight patients with excess fibrotic reaction in the course of diverse diseases were analysed; a tendency that reverted with different IVIg treatment options. Myelofibrosis was predominant in three patients (a patient with a myeloproliferative syndrome, one with systemic lupus erythematosus, and one with Sjogren's syndrome). Three patients had scleroderma as their main feature, one patient had hepatitis C cirrhosis, and one had idiopathic thrombocytopenic purpura. RESULTS: Fibrotic excess was reduced in all the patients by IVIg treatment. In five patients the disease as a whole benefited from the infusion of immunoglobulins. CONCLUSION: IVIg may enhance resorption of fibrosis and promote healing in patients with fibrotic associated disorders.|Aged[MESH]|Female[MESH]|Fibrosis[MESH]|Follow-Up Studies[MESH]|Humans[MESH]|Immunoglobulins, Intravenous/*therapeutic use[MESH]|Male[MESH]|Middle Aged[MESH]|Primary Myelofibrosis/*therapy[MESH]|Rheumatic Diseases/*therapy[MESH]|Scleroderma, Systemic/therapy[MESH]|Skin/*pathology[MESH] |
