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Kallmann syndrome: adhesion, afferents, and anosmia MacColl G; Bouloux P; Quinton RNeuron 2002[May]; 34 (5): 675-8Three new studies into the function of human anosmin-1 and related proteins in C. elegans and rodents show that these influence axon branching and axon targeting. The rodent anosmin appears to work at two stages of development, initially promoting axon outgrowth from the olfactory bulb and then stimulating branching from axons into the olfactory cortex. CeKal-1 further influences morphogenesis, and, as the human and nematode anosmins are functionally conserved, these studies provide insights into the pathogenesis of Kallmann syndrome (KS).|*Extracellular Matrix Proteins[MESH]|Animals[MESH]|Cell Adhesion Molecules/deficiency/genetics[MESH]|Cell Adhesion/genetics[MESH]|Cell Differentiation/*genetics[MESH]|Cell Movement/*genetics[MESH]|Humans[MESH]|Kallmann Syndrome/*genetics/metabolism/physiopathology[MESH]|Nerve Tissue Proteins/deficiency/genetics[MESH]|Olfaction Disorders/*genetics[MESH]|Olfactory Pathways/*abnormalities/metabolism/physiopathology[MESH] |
