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eNOS
LE WE PMID CA
eNOS4061eNOS

Blood vessels (Physiology)

Caveolin and caveoles

EDHF endothelium dependent hyperpolarizing factor

Endothelial dysfunction

eNOS

Insulin (BASKET)

Insulin (resistance)

Lipid rafts and membrane microdomains

Mechanotransduction

Prostacyclin

RAAS (Renin angiotensin system)

2005  
1
Tetrahydrobiopterin and eNOS dimer/monomer ratio--a clue to eNOS uncoupling in diabetes?
[15721855] Cardiovasc Res 65(4): 768-9 (2005)
1999  
2
Signal transduction of eNOS activation.
[10690325] Cardiovasc Res 43(3): 532-41 (1999)
1999  
3
Tetrahydrobiopterin and endothelial nitric oxide synthase activity.
[10536654] Cardiovasc Res 43(2): 274-8 (1999)
2007  
4
Life history of eNOS: partners and pathways.
[17466957] Cardiovasc Res 75(2): 247-60 (2007)
2007  
5
Cardiovascular actions of insulin.
[17525361] Endocr Rev 28(5): 463-91 (2007)
2009  
6
eNOS activation by physical forces: from short-term regulation of contraction to chronic remodeling of cardiovascular tissues.
[19342613] Physiol Rev 89(2): 481-534 (2009)
2011  
7
eNOS activation and NO function: structural motifs responsible for the posttranslational control of endothelial nitric oxide synthase activity.
[21642378] J Endocrinol 210(3): 271-84 (2011)
2011  
8
eNOS activation and NO function: differential control of steroidogenesis by nitric oxide and its adaptation with hypoxia.
[21653733] J Endocrinol 210(3): 259-69 (2011)
2010  
9
Hormonal modulation of endothelial NO production.
[20213497] Pflugers Arch 459(6): 841-51 (2010)
2009  
10
Caveolae, caveolin and control of vascular tone: nitric oxide (NO) and endothelium derived hyperpolarizing factor (EDHF) regulation.
[20083858] J Physiol Pharmacol 60 Suppl 4(): 105-9 (2009)
2007  
11
Interaction of endothelial nitric oxide and angiotensin in the circulation.
[17329548] Pharmacol Rev 59(1): 54-87 (2007)
PMC   
12
H(2)S protects against pressure overload-induced heart failure via upregulation of endothelial nitric oxide synthase.
[23393010] Circulation 127(10):1116-27 (2013)

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All Medline data shown were primarily retrieved from MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.


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