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TRAIL APO2
LE WE PMID CA
TRAIL APO25091TRAIL APO2

Aids

Apoptosis

CD31 PECAM1

CD95 FAS Apo1

CD95L FASL

Death receptor5 DR5

Dendritic cell (Plasmacytoid)

Diabetes mellitus (Involvement kidney)

Interferon beta

Interleukin24 MDA7

Kidney (Disease models)

Neoplasia (Oncolytic virus)

RNA (tRNA)

Skin (Neoplasia Melanoma)

Tolerance (Immunology)

TRAIL APO2

TWEAK Fn14

2005  
1
Current understanding on the immunological functions of tumor necrosis factor-related apoptosis-inducing ligand.
[16274624] Cell Mol Immunol 2(4): 265-9 (2005)
2004  
2
TRAIL and its receptors as targets for cancer therapy.
[15504243] Cancer Sci 95(10): 777-83 (2004)
2003  
3
Apo2L/TRAIL and its death and decoy receptors.
[12655296] Cell Death Differ 10(1): 66-75 (2003)
2010  
4
Cellular dynamics of tRNAs and their genes.
[19931532] FEBS Lett 584(2): 310-7 (2010)
2010  
5
TNF superfamily: a growing saga of kidney injury modulators.
[20953353] Mediators Inflamm 2010(-): - (2010)
2009  
6
Following TRAIL's path in the immune system.
[19476510] Immunology 127(2): 145-54 (2009)
2002  
7
Inhibition of human lung cancer growth following adenovirus-mediated mda-7 gene expression in vivo.
[12085234] Oncogene 21(29): 4558-66 (2002)
2008  
8
Killing of human melanoma cells induced by activation of class I interferon-regulated signaling pathways via MDA-7/IL-24.
[18511292] Cytokine 43(1): 34-44 (2008)
2007  
9
TRAIL in cancer therapy: present and future challenges.
[17907960] Expert Opin Ther Targets 11(10): 1299-314 (2007)
2008  
10
TRAIL receptor-targeted therapeutics: resistance mechanisms and strategies to avoid them.
[18374623] Drug Resist Updat 11(1-2): 17-24 (2008)
2009  
11
TNF-related apoptosis-inducing ligand (TRAIL): a new path to anti-cancer therapies.
[19836385] Eur J Pharmacol 625(1-3): 63-72 (2009)
2007  
12
HIV-1 immunopathogenesis: how good interferon turns bad.
[17112786] Clin Immunol 123(2): 121-8 (2007)

Downloaded from http://www.kidney.de - The Database of Free Medical Reviews - The Metatextbook of Medicine - Ossip Groth

All Medline data shown were primarily retrieved from MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.


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