LE WE PMID CA
SLC26A4 Pendrin6348SLC26A4 Pendrin

Calcium (Hypercalciuria)

Calcium (Physiology renal)

Hearing (Neurophysiology)

NCX1

NIS

Pendred s syndrome

SLC26A1

SLC26A2

SLC26A3

SLC26A4 Pendrin

SLC26A6

SLC26A7

SLC26A9

Thyroid (Physiology)

Transporter (Chloride and bicarbonate)

Transporter (Sodium and iodine)

TRPV5

TRPV6

2007  
1
The influence of mutations in the SLC26A4 gene on the temporal bone in a population with enlarged vestibular aqueduct.
[17309986] Arch Otolaryngol Head Neck Surg 133(2): 162-8 (2007)
2009  
2
Diverse transport modes by the solute carrier 26 family of anion transporters.
[19015189] J Physiol 587(Pt 10): 2179-85 (2009)
2009  
3
Minireview: The sodium-iodide symporter NIS and pendrin in iodide homeostasis of the thyroid.
[19196800] Endocrinology 150(3): 1084-90 (2009)
2007  
4
Loss of cochlear HCO3- secretion causes deafness via endolymphatic acidification and inhibition of Ca2+ reabsorption in a Pendred syndrome mouse model.
[17299139] Am J Physiol Renal Physiol 292(5): F1345-53 (2007)
2007  
5
Lack of pendrin HCO3- transport elevates vestibular endolymphatic [Ca2+] by inhibition of acid-sensitive TRPV5 and TRPV6 channels.
[17200157] Am J Physiol Renal Physiol 292(5): F1314-21 (2007)
2012  
6
Deletion of the Cl-/HCO3- exchanger pendrin downregulates calcium-absorbing proteins in the kidney and causes calcium wasting.
[21873623] Nephrol Dial Transplant 27(4): 1368-79 (2012)

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  • SLC26A4 Pendrin

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